
Chapter
1 . Basic and special concepts of biomechanics
Dr. Croft
begins with a discussion of the properties of discs, ligaments,
muscle and bone, as well as the normal motion of the cervical
spine. Soft tissues have viscoelastic material properties
which is key to understanding injury mechanisms. He will
also touch on the concepts of coupled, intersegmental, and
paradoxic motion.
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Chapter
2. Injury impairment scales (AIS, IIS, ISS, KABCO)
Dr. Croft will explain the now widely
adopted cervical acceleration/deceleration (CAD) (or whiplash-associated
disorder or WAD) grading system which he developed more than a
decade ago. This grading system, which has now been validated in
numerous studies, provides the basis for effective intervention
and for prognostication.
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Chapter
3. Epidemiology of whiplash
Dr. Croft will explain the tremendous
scope of the current public health problem and the factors contributing
to
it. He reviews the substantial international literature, including
the latest crash data of real world crashes obtained from on-board
black boxes. He develops from this analysis—as well as his own
published research—a risk estimate for adults and for children,
incidence rates, and the prevalence of chronic pain in the U.S. attributable
to motor vehicle trauma. With all the confusion and misinformation
that exist regarding low speed crashes, amounts of property damage,
and relative risk, Dr. Croft is careful to clarify these issues in
great detail. For example, and perhaps counter-intuitively, within
a specific range of low speed crashes, it is shown that the risk
for occupant injury is actually greater when property damage is minimal.
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Chapter
4. Brain, neck, and cervical spine trauma mechanisms from motor
vehicle crashes (MVC)
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The discussion begins with an introduction
of the nomenclature and progresses to a discussion of velocity
change (delta V), barrier velocity, and the ways in which velocity,
time, and acceleration interact to affect the risk for occupant
injury. Dr. Croft then takes attendees on an exploration of our
current knowledge based on mathematical models, animal experiments
(including the porcine experiments from whence the neck injury
criterion (NIC) was proposed), cadaver experiments (where many
recent discoveries have been made), dummy experiments (including
those of the newest rear impact dummies or RID, which have been
tested at the Spine Research Institute of San Diego by Dr. Croft
and others), and, finally, the numerous human subject volunteer
crash tests. Dr. Croft discusses his several years of crash testing
as well as all of the other serious research in that area. As always,
his presentation is replete with video footage and illustration
to assist in understanding. Also in this section, Dr. Croft will
discuss crash vectors other than rear, the important vehicle and
subject parameters which modify risk, the dubious practice of estimating
injury risk from property damage, the New Car Assessment Program
and its unintended effects in low speed crashes, head restraints,
seat backs, air bags, and other safety systems and how they modify
injury risk and severity. Also discussed will be the general sequence
of kinematic events in CAD injury and their relative timing, the
head injury criterion (HIC), the neck injury criterion (NIC), and
other important injury assessment reference values (IARV).
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Chapter
5. Soft tissue injuries
This section begins with a discussion
of soft and hard tissue injuries resulting from whiplash, including
data obtained from experimental animal research and surgical and
autopsy findings. Fractures (stable and unstable), dislocations,
and the broad range of soft tissue lesions, or WAD, will be discussed
in the context of mechanism of injury and vector-based occupant
kinematics. Dr. Croft has catalogued an immense database of this
literature. He will offer an explanation for most of the common
components of CAD/WAD including headache, various types of neck
and back pain, shoulder and upper extremity pain, lower extremity
pain, neurological syndromes, dizziness/lightheadedness, vertigo,
visual disturbances, cognitive, and endocrinological disorders,
and will additionally explore the common phenomenon of delayed
onset of symptomatology.
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Chapter
6. Common syndromes
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Common syndromes associated with whiplash
include cognitive disorders, usually resulting from mild traumatic
brain injury (MTBI), postconcussion syndrome (PCS), thoracic outlet
syndrome (TOS), TM joint disorder, carpal tunnel syndrome (CTS),
posttraumatic headache, myofascitis, and numerous other less common
conditions. Also discussed will be the clinical and diagnostic
components of each, again making constant reference to current
world literature. Common symptoms of whiplash will be explained
and, for those conditions that remain poorly understood, Dr. Croft
will discuss the relevant literature and develop and/or describe
the best and most current hypotheses. Dr. Croft goes into extensive
detail on the subject of MTBI, ranging from the history of our
knowledge base to current predictive models, and outlines the range
of common symptoms resulting from this all too common, but poorly
understood, condition. Headaches are discussed in the context of
current classification systems, including that of the International
Headache Society. Long-term consequences of MTBI in children and
adults is also discussed.
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Chapter
7. Conditions affecting muscles
Dr. Croft will discuss the two most
common forms of posttraumatic muscle disorder: myofascitis and
fibromyalgia, going into detail about their respective diagnostic
methods and criteria, such as those of the American College of
Rheumatology. He will also provide a historical account of this
controversial area, beginning with Virchow in 1852, and ending
with the latest research findings.
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Chapter
8. Pain syndromes
Neurogenic pain, i.e., the pain that
is conducted from peripheral nociceptor to dorsal root ganglion,
to spinal cord, and eventually to the somatosensory cortex via
the lateral spinothalamic tracts and thalamus, is the type studied
and understood by most practitioners. However, few practitioners
possess a deep understanding of discogenic, vertebrogenic, and
scleragenic pain mechanisms, all of which are critically important
in the diagnosis, management, and medicolegal explanation of most
CAD cases. Dr. Croft explores the various pain mechanisms associated
with CAD injury and its sequelae. He also looks at the epidemiology
of chronic pain, sleep disturbance, and depression as an alternate
explanation for some CAD symptoms.
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Chapter
9. Outcome of CAD injury
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For many,
this is perhaps the most poorly understood area of this science,
despite the magnitude of the current literature that is available.
It is also one of the most hotly contested subjects from a forensic
standpoint. Yet, in spite of the commonly voiced misconceptions
about recovery from CAD, the preponderance of the evidence is clear
and unambiguous. Dr. Croft reviews the substantial outcome literature
and statistics in detail, and presents not only a thorough meta-analysis
of it, but also develops a comprehensive risk analysis methodology
found nowhere else. He explores the known risk factors for acute
injury, as well as the risk factors for poor outcome—information
that provides the physician and the patient with a meaningful guide
to management and a scientific basis for clinical expectation.
Moreover, Dr. Croft will emphasize the unique qualifications and
role of the physician in this comprehensive risk analysis, which
typically overshadows simple crash reconstruction or biomechanical
analysis in its scope and construct validity. The section concludes
with a review of the current literature on litigation neurosis
concerning CAD and MTBI.
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Spine Research Institute of San Diego, Inc.
826 Orange Avenue, Suite 633
Coronado, CA 92118
USA
Voice: (619) 423-9867 or (619) 423-5475
Monday-Friday 9:00 am- 5:00 pm (PST)
Fax: (619) 423-3084
Email: info@srisd.com |